Despite the progressive closure of large collieries, silicosis will
remain a topical issue for many years to, come. However, given the scattering
and small size of companies exposing their workers to siliceous dusts,
knowledge on this pathology is likely to become dispersed and decline
in the long run.
Apart from. the few cases of silicosis that become acute generally
because of massive exposure, the labour world will know only about the
milder forras of the disease by the time people retire.
The organisation of post-occupational monitoring in the former coal
basin of the Nord Pas de Calais a real observatory of pensioners sufféring
from silicosis has led to an awareness of the evolutionary potential
of this disease which leads sooner or later to complete destruction
of the lungs.
The various therapeutic tests such as the administration of aerosols
of aluminium salts have not allowed this evolution to be stopped. However
we have been able to observe a significant decrease in the number of
silicoses, that could have become acute, in former miners removed from.
risk exposure at an early stage as soon as the first signs of the disease
appeared. Also, the later the first radiological signs appear, the lower
the evolution from the milder to the more acute forms of the disease.
For want of a curative therapy, prevention remains the master word.
It should be applied at all levels: industrial by reducing dust levels,
medical by the earliest exit of persons presenting radiological signs
of a pulmonary disease.
The past fifty years have however seen therapeutic progress, particularly
regarding the management of respiratory insufficiency and its complications.
However, a lung that bas been destroyed does not regenerate and the
only solution consists in making best use of what remains.
To be effective this action should be undertaken early with active
participation of patients. It firstly requires an improvement in health
practices (suppression of tobacco, keeping of a physical activity, combating
obesity, etc. ...).
Respiratory reeducation and effort re-training under the guidance of
a physiotherapist can delay the appearance of a serious respiratory
insufficiency and preserve autonomy to the maximum. They can be started
during a cure at a spa but should be continued when back at home.
Even in the most serious cases, requiring long-term oxygenotherapy,
autonomy should remain the therapist's first concern. Oxygenotherapy
is now facilitated by the use of ambulatory equipment using liquid oxygen.
Lastly, treatment for sufférers of very serious respiratory
insufficiency can now be given in the family setting by using ventilation
equipment, putting an end to isolation in hospitals.
This appraisal may appear sombre but the management of the problem,
currently facilitated by the geographic concentration of the affected
population, has allowed effective treatinent protocols to be developed.
Let's hope that these bases contribute to, the continuation of equally
effective treatinent for the isolated cases who are going to become
the majority.